4 edition of Placental Signals Autocrine and Paracine Control of Pregnancy (Trophoblast Research) found in the catalog.
Placental Signals Autocrine and Paracine Control of Pregnancy (Trophoblast Research)
February 11, 1993
by University of Rochester Press
Written in English
|Contributions||Lise Cedard (Editor), Anthony Firth (Editor)|
|The Physical Object|
|Number of Pages||400|
In rat placenta, which is structurally different from human placenta, both Kiss-1 and its receptor are predominantly expressed by the invasive trophoblast giant cells, suggesting autocrine control. The placenta is considered an important organ that evolves with the implantation of the blastocyst throughout the pregnancy. The placenta has an essential role in functions such as nutrition, excretion, and immunologic and endocrine function. The normal placenta is a round- or oval-shaped organ that attaches to the uterine wall and has roughly 22 cm in diameter and a thickness of about 2– Cited by: 1.
Thus, trophoblast and endothelial coexpression of SLIT/ROBO suggests an autocrine/paracrine regulatory system for regulating placental function. Differential expression of SLITs and ROBOs in healthy term and preeclamptic placentas and hypoxia regulation of their expressions in placental cells implicate a potential pathophysiological role for Cited by: Placental Lactogen. Placental lactogens are Type I cytokines produced by the placenta during pregnancy that share structural homology with PRL and GH and can also influence pregnancy-related mammary development (Forsyth, ). the locally-induced IGF-I will act as an autocrine and paracrine growth factor.
Cell Host & Microbe Short Article Type III Interferons Produced by Human Placental Trophoblasts Confer Protection against Zika Virus Infection Avraham Bayer,1,2,7 Nicholas J. Lennemann,3,7 Yingshi Ouyang,1,2 John C. Bramley,3 Stefanie Morosky,3 Ernesto Torres De Azeved Marques, Jr.,4,5 Sara Cherry,6 Yoel Sadovsky, 1,2 3* and Carolyn B. Coyne * 1Magee-Womens Research Institute, . PDF | Diabetes in pregnancy is associated with a derangement of hormones, cytokines, metabolites and growth factors in the maternal and foetal | Find, read and cite all the research you need on.
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Fant M, Munro H, and Moses AC, An autocrine/paracrine role for insulin-like growth factors in the regulation of human placental growth. J Clin Endocrinol Metab (), – Cited by: 1. Based on the major role played by the human placenta in pregnancy and the cascade of labor processes activated via placental mediators exerting endocrine, paracrine, and autocrine actions, this review article has aimed at presenting the role of these mediators in term and preterm labor and the molecular pathways of their actions.
some of the aforementioned mediators are Cited by: Introduction: brain and placenta, birth and behavior, health and disease into the regulation and control of human pregnancy. A subtext of the book is the They act as endocrine, paracrine, and autocrine factors, to control the secretion of.
Studies on placental physiology have offered a model of interaction within and between intrauterine tissues through cell-to-cell communication (paracrine) or/and within the same cells (autocrine).
The network of peptides expressed in the human placenta are involved in regulating the feto-maternal by: Placental lactogen (PL) is produced only during pregnancy and may be one of the endocrine factors that controls mammary growth.
It is also generally recognized that mammary growth is controlled not only by endocrine signals, but also by paracrine and/or autocrine by: 8.
Endocrine, paracrine, and autocrine placental mediators in labor Article (PDF Available) in Hormones (Athens, Greece) 11(4) October with Reads How we measure 'reads'. The LHCGR present in mural cells is responsible for generating the signals that trigger the paracrine and autocrine pathways that lead to ovulation.
For the purposes of this review, ovulation is divided into three discreet steps: resumption of meiosis, expansion of cumulus cells, and follicular rupture (Figure 1).Cited by: The authors hypothesized that GDM modulates paracrine regulation of feto-placental angiogenesis by the trophoblast and investigated the composition Cited by: 8.
87 placenta is, therefore, exposed to hormones by autocrine, paracrine and endocrine 88 mechanisms from early in development. However, compared to the fetus, less is known 89 about the role of endocrine signals in placental development (2). This review, therefore, 90 examines the endocrine regulation of placental phenotype.
It places particular. The placenta is, therefore, exposed to hormones by autocrine, paracrine and endocrine mechanisms from early in development. However, compared to the fetus, less is known about the role of endocrine signals in placental development.
This review, therefore, examines the endocrine regulation of placental by: The endocrinology of human pregnancy is dominated by multiple placental hormones that induce physiologic changes in the mother in favor of pregnancy.
Hormones synthesized by the placenta include chorionic gonadotropin which prevents regression of the ovarian corpus luteum during the first several weeks of pregnancy and progesterone which. REVIEW The endocrine function of human placenta: an overview Mariana A Costa *,1 Faculdade de Farmácia, Universidade do Porto, Porto, Portugal * E-mail address: [email protected] 1 Mariana A Costa obtained her PhD degree in Pharmaceutical Sciences at Faculdade de Farmácia Universidade do Porto, though she is not currently linked to this institution.
Meeting,(3rd: Dourdan, France) Title(s): Placental signals: autocrine and paracine control of pregnancy/ edited by Lise Cedard and Anthony Firth. Country of Publication: United States Publisher: Rochester, N.Y.: University of Rochester Press Other tissues and cells may also produce 1,25(OH) 2 D for local autocrine/paracrine effects.
19 For example, during pregnancy, the placenta expresses both the vitamin D receptor and the vitamin D. PDF | The placenta acts as a major organ for hematopoiesis. It is believed that placental hematopoietic stem and progenitor cells (HSPCs) migrate to the | Find, read and cite all the research.
Placental development and function are tightly regulated by endocrine, paracrine, and autocrine factors present in the maternal and the fetal circulation and in the placenta.
Gestational diabetes (GDM) is associated with a derangement of the concentration of several hormones, cytokines, metabolites, and growth factors in both circulations that Cited by: 3.
The trophoblast compartment of the placenta comprises various subpopulations with distinct functions. They interact among each other by secreted signals thus forming autocrine or paracrine regulatory by: Abstract.
During the 9-month time period of human pregnancy, the placenta is a pleiotropic regulator of the intrauterine environment. The principle cell type of the placenta, trophoblast, produces a wide variety of hormones that signal maternal, placental, and fetal tissues in an autocrine, paracrine.
Placental STAT3 signaling is activated but mTOR signaling is unaffected in PCOS. WHAT IS KNOWN ALREADY Women with PCOS have increased risk of poor pregnancy outcomes (e.g. restricted or accelerated fetal growth), indicating placental by:. Click Here ?book=XDownload Human Placental Trophoblasts: Impact of Maternal Nutrition Popular Books.The regulatory mechanism(s) for the production of human placental peptide hormones are poorly understood and two theories currently prevail: an autonomous production which varies according to the changing ratio of cyto- to syncytiotrophoblasts throughout pregnancy an autocrine-paracrine control mediated through receptor binding by: 8.lntcr (), 12, 3.
Regulation of the Synthesis and Release of Decidual Prolactin by Placental and Autocrine/Paracrine-Factors STUART HANDWERGER, EDITH MARKOFF & RANDALL RICHARDS Perinatal Research Institute, University yf Cincinnati and Division of Endocrkologv, Childrens Hospital Medical Center, Cincinnuti, OhioUSA Supported by NIH Grant HD *First Cited by: